Samuel A. Profile

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kisumu,

kenya

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Ble med juli 3, 2015

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Samuel A.

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kisumu,

kenya

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Gjenansettelsesrate

Biochemical, Molecular and Data Processing Expert

I hold a Doctor of Philosophy Degree (Ph.D.) in Medical Biochemistry, with over 6 years experience in Biochemical and genetic analysis techniques. I have extensive hands on experience in molecular techniques, cloning, enzyme immuno-assays, nucleic and protein analysis techniques, tissue culture techniques, flow cytometric analysis and immunoflourescence staining techniques. In addition, i specialize bioinformatics, data processing and statistical analysis techniques. I also write and review scientific articles meant for publication in International peer reviewed Journals.

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Utdanning

Ph.D - Medical Biochemistry

Kenyatta University,

Kenya

2007 - 2012

(5 år)

Publikasjoner

Functional Promoter Haplotypes of Interleukin-18 Condition Susceptibility to Severe Malarial Anemia.

Infect. Immun.

Interleukin-18 (IL-18) is important for regulating innate and acquired immunity in inflammatory and infectious diseases. Analysis of two functional SNPs in the IL-18 promoter (−137G→C [rs187238] and −607C→A [rs1946518]) whose haplotypes encompass genetic variation due to the presence of strong linkage disequilibrium among the variants was done. IL-18 promoter haplotypes that condition elevated IL-18 gene products during acute infection are associated with increased risk of severe malaria.

Suppressed circulating bicyclo-PGE2 levels and leukocyte COX-2 transcripts in children co-infected..

Biochem Biophys Res Commun

In holoendemic Plasmodium falciparum transmission regions, malarial anemia is a leading cause of childhood morbidity and mortality. Identifying biomarkers of malaria disease severity is important for understanding the molecular pathways that influence clinical outcomes. Decreased plasma bicyclo-PGE2/creatinine levels and peripheral blood COX-2 transcripts were observed in children with malaria, suggesting that bicyclo-PGE2 may represent both a marker and mediator of malaria pathogenesis.